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PEX5 (NM_001131026) Human Tagged ORF Clone Lentiviral Particle
CAT＃: RC226052L4V
- LentiORF®
-
Lenti ORF particles, PEX5 (mGFP-tagged)-Human peroxisomal biogenesis factor 5 (PEX5), transcript variant 5, 200ul, >10^7 TU/mL
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CNY 12,350.00
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CNY 1,999.00
CNY 2,700.00
Specifications
| Product Data | |
| Product Name | PEX5 (NM_001131026) Human Tagged ORF Clone Lentiviral Particle |
| Synonyms | PBD2A; PBD2B; PTS1-BP; PTS1R; PXR1; RCDP5 |
| Vector | pLenti-C-mGFP-P2A-Puro |
| ACCN | NM_001131026 |
| ORF Size | 1917 bp |
| Sequence Data |
The ORF insert of this clone is exactly the same as(RC226052).
|
| OTI Disclaimer | The molecular sequence of this clone aligns with the gene accession number as a point of reference only. However, individual transcript sequences of the same gene can differ through naturally occurring variations (e.g. polymorphisms), each with its own valid existence. This clone is substantially in agreement with the reference, but a complete review of all prevailing variants is recommended prior to use. More info |
| OTI Annotation | This clone was engineered to express the complete ORF with an expression tag. Expression varies depending on the nature of the gene. |
| Reference Data | |
| RefSeq | NM_001131026.1 |
| RefSeq Size | 2693 bp |
| RefSeq ORF | 1920 bp |
| Locus ID | 5830 |
| Protein Families | Druggable Genome |
| MW | 70.9 kDa |
| Gene Summary | The product of this gene binds to the C-terminal PTS1-type tripeptide peroxisomal targeting signal (SKL-type) and plays an essential role in peroxisomal protein import. Peroxins (PEXs) are proteins that are essential for the assembly of functional peroxisomes. The peroxisome biogenesis disorders (PBDs) are a group of genetically heterogeneous autosomal recessive, lethal diseases characterized by multiple defects in peroxisome function. The peroxisomal biogenesis disorders are a heterogeneous group with at least 14 complementation groups and with more than 1 phenotype being observed in cases falling into particular complementation groups. Although the clinical features of PBD patients vary, cells from all PBD patients exhibit a defect in the import of one or more classes of peroxisomal matrix proteins into the organelle. Defects in this gene are a cause of neonatal adrenoleukodystrophy (NALD), a cause of Zellweger syndrome (ZWS) as well as may be a cause of infantile Refsum disease (IRD). Alternatively spliced transcript variants encoding different isoforms have been identified. [provided by RefSeq, Oct 2008] |
Documents
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