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TSG101 (NM_006292) Human Tagged ORF Clone Lentiviral Particle
CAT＃: RC200275L1V
- LentiORF®
-
Lenti ORF particles, TSG101 (Myc-DDK tagged) - Human tumor susceptibility gene 101 (TSG101), 200ul, >10^7 TU/mL
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CNY 8,930.00
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Specifications
| Product Data | |
| Product Name | TSG101 (NM_006292) Human Tagged ORF Clone Lentiviral Particle |
| Synonyms | TSG10; VPS23 |
| Vector | pLenti-C-Myc-DDK |
| ACCN | NM_006292 |
| ORF Size | 1170 bp |
| Sequence Data |
The ORF insert of this clone is exactly the same as(RC200275).
|
| OTI Disclaimer | The molecular sequence of this clone aligns with the gene accession number as a point of reference only. However, individual transcript sequences of the same gene can differ through naturally occurring variations (e.g. polymorphisms), each with its own valid existence. This clone is substantially in agreement with the reference, but a complete review of all prevailing variants is recommended prior to use. More info |
| OTI Annotation | This clone was engineered to express the complete ORF with an expression tag. Expression varies depending on the nature of the gene. |
| Reference Data | |
| RefSeq | NM_006292.2 |
| RefSeq Size | 1562 bp |
| RefSeq ORF | 1173 bp |
| Locus ID | 7251 |
| Domains | UBCc |
| Protein Families | Druggable Genome, Transcription Factors |
| Protein Pathways | Endocytosis |
| MW | 43.9 kDa |
| Gene Summary | The protein encoded by this gene belongs to a group of apparently inactive homologs of ubiquitin-conjugating enzymes. The gene product contains a coiled-coil domain that interacts with stathmin, a cytosolic phosphoprotein implicated in tumorigenesis. The protein may play a role in cell growth and differentiation and act as a negative growth regulator. In vitro steady-state expression of this tumor susceptibility gene appears to be important for maintenance of genomic stability and cell cycle regulation. Mutations and alternative splicing in this gene occur in high frequency in breast cancer and suggest that defects occur during breast cancer tumorigenesis and/or progression. [provided by RefSeq, Jul 2008] |
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