Mlycd (NM_019966) Mouse Tagged ORF Clone Lentiviral Particle
CAT#: MR207886L3V
- LentiORF®
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Lenti ORF particles, Mlycd (Myc-DDK-tagged) - Mouse malonyl-CoA decarboxylase (Mlycd), nuclear gene encoding mitochondrial protein, 200ul, >10^7 TU/mL
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CNY 9,405.00
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Specifications
Product Data | |
Product Name | Mlycd (NM_019966) Mouse Tagged ORF Clone Lentiviral Particle |
Synonyms | AI324784; Mcd |
Vector | pLenti-C-Myc-DDK-P2A-Puro |
ACCN | NM_019966 |
ORF Size | 1476 bp |
Sequence Data |
The ORF insert of this clone is exactly the same as(MR207886).
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OTI Disclaimer | The molecular sequence of this clone aligns with the gene accession number as a point of reference only. However, individual transcript sequences of the same gene can differ through naturally occurring variations (e.g. polymorphisms), each with its own valid existence. This clone is substantially in agreement with the reference, but a complete review of all prevailing variants is recommended prior to use. More info |
OTI Annotation | This clone was engineered to express the complete ORF with an expression tag. Expression varies depending on the nature of the gene. |
Reference Data | |
RefSeq | NM_019966.2 |
RefSeq Size | 2116 bp |
RefSeq ORF | 1479 bp |
Locus ID | 56690 |
Gene Summary | Catalyzes the conversion of malonyl-CoA to acetyl-CoA. In the fatty acid biosynthesis MCD selectively removes malonyl-CoA and thus assures that methyl-malonyl-CoA is the only chain elongating substrate for fatty acid synthase and that fatty acids with multiple methyl side chains are produced. In peroxisomes it may be involved in degrading intraperoxisomal malonyl-CoA, which is generated by the peroxisomal beta-oxidation of odd chain-length dicarboxylic fatty acids. Plays a role in the metabolic balance between glucose and lipid oxidation in muscle independent of alterations in insulin signaling. Plays a role in controlling the extent of ischemic injury by promoting glucose oxidation.[UniProtKB/Swiss-Prot Function] |
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