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FANCA (NM_001018112) Human Tagged ORF Clone Lentiviral Particle
CAT＃: RC223218L3V
- LentiORF®
-
Lenti ORF particles, FANCA (Myc-DDK tagged) - Human Fanconi anemia, complementation group A (FANCA), transcript variant 2, 200ul, >10^7 TU/mL
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CNY 8,170.00
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Specifications
| Product Data | |
| Product Name | FANCA (NM_001018112) Human Tagged ORF Clone Lentiviral Particle |
| Synonyms | FA; FA-H; FA1; FAA; FACA; FAH; FANCH |
| Vector | pLenti-C-Myc-DDK-P2A-Puro |
| ACCN | NM_001018112 |
| ORF Size | 891 bp |
| Sequence Data |
The ORF insert of this clone is exactly the same as(RC223218).
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| OTI Disclaimer | The molecular sequence of this clone aligns with the gene accession number as a point of reference only. However, individual transcript sequences of the same gene can differ through naturally occurring variations (e.g. polymorphisms), each with its own valid existence. This clone is substantially in agreement with the reference, but a complete review of all prevailing variants is recommended prior to use. More info |
| OTI Annotation | This clone was engineered to express the complete ORF with an expression tag. Expression varies depending on the nature of the gene. |
| Reference Data | |
| RefSeq | NM_001018112.1 |
| RefSeq Size | 1673 bp |
| RefSeq ORF | 894 bp |
| Locus ID | 2175 |
| Protein Families | Druggable Genome |
| MW | 32.8 kDa |
| Gene Summary | The Fanconi anemia complementation group (FANC) currently includes FANCA, FANCB, FANCC, FANCD1 (also called BRCA2), FANCD2, FANCE, FANCF, FANCG, FANCI, FANCJ (also called BRIP1), FANCL, FANCM and FANCN (also called PALB2). The previously defined group FANCH is the same as FANCA. Fanconi anemia is a genetically heterogeneous recessive disorder characterized by cytogenetic instability, hypersensitivity to DNA crosslinking agents, increased chromosomal breakage, and defective DNA repair. The members of the Fanconi anemia complementation group do not share sequence similarity; they are related by their assembly into a common nuclear protein complex. This gene encodes the protein for complementation group A. Alternative splicing results in multiple transcript variants encoding different isoforms. Mutations in this gene are the most common cause of Fanconi anemia. [provided by RefSeq, Jul 2008] |
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