Lgmn (NM_011175) Mouse Tagged ORF Clone Lentiviral Particle
CAT#: MR221565L3V
- LentiORF®
-
Lenti ORF particles, Lgmn (Myc-DDK-tagged) - Mouse legumain (Lgmn), 200ul, >10^7 TU/mL
Need custom lentivirus service?
Get a free quote
CNY 8,740.00
货期*
详询
规格
Product images
![](https://cdn.origene.com/img/defaults-img.jpg)
经常一起买 (2)
Specifications
Product Data | |
Product Name | Lgmn (NM_011175) Mouse Tagged ORF Clone Lentiviral Particle |
Synonyms | A; AEP; AI746452; AU022324; Pr; Prsc1 |
Vector | pLenti-C-Myc-DDK-P2A-Puro |
ACCN | NM_011175 |
ORF Size | 1308 bp |
Sequence Data |
The ORF insert of this clone is exactly the same as(MR221565).
|
OTI Disclaimer | The molecular sequence of this clone aligns with the gene accession number as a point of reference only. However, individual transcript sequences of the same gene can differ through naturally occurring variations (e.g. polymorphisms), each with its own valid existence. This clone is substantially in agreement with the reference, but a complete review of all prevailing variants is recommended prior to use. More info |
OTI Annotation | This clone was engineered to express the complete ORF with an expression tag. Expression varies depending on the nature of the gene. |
Reference Data | |
RefSeq | NM_011175.2 |
RefSeq Size | 1874 bp |
RefSeq ORF | 1308 bp |
Locus ID | 19141 |
Gene Summary | This gene encodes a member of the cysteine peptidase family C13 that plays an important role in the endosome/lysosomal degradation system. The encoded inactive preproprotein undergoes autocatalytic removal of the C-terminal inhibitory propeptide to generate the active endopeptidase that cleaves protein substrates on the C-terminal side of asparagine residues. Mice lacking the encoded protein exhibit defects in the lysosomal processing of proteins resulting in their accumulation in the lysosomes, and develop symptoms resembling hemophagocytic lymphohistiocytosis. [provided by RefSeq, Aug 2016] |
Documents
Product Manuals |
FAQs |
SDS |
Resources
You may also need
Customer
Reviews
Loading...