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Ndufv1 (NM_133666) Mouse Tagged ORF Clone Lentiviral Particle
CAT＃: MR207415L3V
- LentiORF®
-
Lenti ORF particles, Ndufv1 (Myc-DDK-tagged) - Mouse NADH dehydrogenase (ubiquinone) flavoprotein 1 (Ndufv1), nuclear gene encoding mitochondrial protein, 200ul, >10^7 TU/mL
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CNY 9,120.00
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Specifications
| Product Data | |
| Product Name | Ndufv1 (NM_133666) Mouse Tagged ORF Clone Lentiviral Particle |
| Synonyms | CI-51kD |
| Vector | pLenti-C-Myc-DDK-P2A-Puro |
| ACCN | NM_133666 |
| ORF Size | 1395 bp |
| Sequence Data |
The ORF insert of this clone is exactly the same as(MR207415).
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| OTI Disclaimer | The molecular sequence of this clone aligns with the gene accession number as a point of reference only. However, individual transcript sequences of the same gene can differ through naturally occurring variations (e.g. polymorphisms), each with its own valid existence. This clone is substantially in agreement with the reference, but a complete review of all prevailing variants is recommended prior to use. More info |
| OTI Annotation | This clone was engineered to express the complete ORF with an expression tag. Expression varies depending on the nature of the gene. |
| Reference Data | |
| RefSeq | NM_133666.2 |
| RefSeq Size | 1608 bp |
| RefSeq ORF | 1395 bp |
| Locus ID | 17995 |
| Gene Summary | This gene encodes a subunit of the NADH-ubiquinone oxidoreductase (complex I) enzyme, which is a large, multimeric protein. It is the first enzyme complex in the mitochondrial electron transport chain and catalyzes the transfer of electrons from NADH to the electron acceptor ubiquinone. The proton gradient created by electron transfer drives the conversion of ADP to ATP. This gene is a core subunit and is conserved in prokaryotes and eukaryotes. The human ortholog of this protein has been characterized. It has consensus motifs for NADH, flavin mononucleotide, and iron-sulfur binding sites and participates in the oxidation of NADH as part of the dehydrogenase module of complex I. In humans, deficiencies in complex I are associated with myopathies, encephalomyopathies, and neurodegenerative disorders. [provided by RefSeq, Jun 2013] |
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