FGF 23 (FGF23) (NM_020638) Human Tagged ORF Clone Lentiviral Particle

CAT#: RC210127L3V

  • LentiORF®

Lenti ORF particles, FGF23 (Myc-DDK tagged) - Human fibroblast growth factor 23 (FGF23), 200ul, >10^7 TU/mL

ORF Plasmid: DDK tGFP



Need custom lentivirus service?
Get a free quote

CNY 7,410.00


货期*
详询

规格
    • 200 ul

Product images

经常一起买 (3)
Lenti ORF control particles of pLenti-C-Myc-DDK-P2A-Puro, >1x10^7 TU/ml, 0.5 ml
    • 500 ul

CNY 2,950.00


One-Wash Lentivirus Titer Kit, HIV-1 p24 ELISA
    • 96 reactions

CNY 5,230.00


FGF23 Rabbit monoclonal antibody,clone OTIR5D11
    • 100 ul

CNY 1,999.00
CNY 4,070.00

Specifications

Product Data
Product Name FGF 23 (FGF23) (NM_020638) Human Tagged ORF Clone Lentiviral Particle
Synonyms ADHR; FGFN; HFTC2; HPDR2; HYPF; PHPTC
Vector pLenti-C-Myc-DDK-P2A-Puro
ACCN NM_020638
ORF Size 753 bp
Sequence Data
The ORF insert of this clone is exactly the same as(RC210127).
OTI Disclaimer The molecular sequence of this clone aligns with the gene accession number as a point of reference only. However, individual transcript sequences of the same gene can differ through naturally occurring variations (e.g. polymorphisms), each with its own valid existence. This clone is substantially in agreement with the reference, but a complete review of all prevailing variants is recommended prior to use. More info
OTI Annotation This clone was engineered to express the complete ORF with an expression tag. Expression varies depending on the nature of the gene.
Reference Data
RefSeq NM_020638.2
RefSeq Size 3018 bp
RefSeq ORF 756 bp
Locus ID 8074
Protein Families Druggable Genome, Secreted Protein
Protein Pathways MAPK signaling pathway, Melanoma, Pathways in cancer, Regulation of actin cytoskeleton
MW 28 kDa
Gene Summary This gene encodes a member of the fibroblast growth factor family of proteins, which possess broad mitogenic and cell survival activities and are involved in a variety of biological processes. The product of this gene regulates phosphate homeostasis and transport in the kidney. The full-length, functional protein may be deactivated via cleavage into N-terminal and C-terminal chains. Mutation of this cleavage site causes autosomal dominant hypophosphatemic rickets (ADHR). Mutations in this gene are also associated with hyperphosphatemic familial tumoral calcinosis (HFTC). [provided by RefSeq, Feb 2013]
*Delivery time may vary from web posted schedule. Occasional delays may occur due to unforeseen complexities in the preparation of your product. International customers may expect an additional 1-2 weeks in shipping.
Customer Reviews 
Loading...