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Angiotensinogen (AGT) (NM_000029) Human Tagged ORF Clone Lentiviral Particle
CAT＃: RC203768L4V
- LentiORF®
-
Lenti ORF particles, AGT (mGFP-tagged) - Human angiotensinogen (serpin peptidase inhibitor, clade A, member 8) (AGT), 200ul, >10^7 TU/mL
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CNY 9,975.00
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Specifications
| Product Data | |
| Product Name | Angiotensinogen (AGT) (NM_000029) Human Tagged ORF Clone Lentiviral Particle |
| Synonyms | ANHU; hFLT1; SERPINA8 |
| Vector | pLenti-C-mGFP-P2A-Puro |
| ACCN | NM_000029 |
| ORF Size | 1455 bp |
| Sequence Data |
The ORF insert of this clone is exactly the same as(RC203768).
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| OTI Disclaimer | The molecular sequence of this clone aligns with the gene accession number as a point of reference only. However, individual transcript sequences of the same gene can differ through naturally occurring variations (e.g. polymorphisms), each with its own valid existence. This clone is substantially in agreement with the reference, but a complete review of all prevailing variants is recommended prior to use. More info |
| OTI Annotation | This clone was engineered to express the complete ORF with an expression tag. Expression varies depending on the nature of the gene. |
| Reference Data | |
| RefSeq | NM_000029.2 |
| RefSeq Size | 2587 bp |
| RefSeq ORF | 1458 bp |
| Locus ID | 183 |
| Domains | SERPIN |
| Protein Families | Druggable Genome, Secreted Protein |
| Protein Pathways | Renin-angiotensin system |
| MW | 53.1 kDa |
| Gene Summary | The protein encoded by this gene, pre-angiotensinogen or angiotensinogen precursor, is expressed in the liver and is cleaved by the enzyme renin in response to lowered blood pressure. The resulting product, angiotensin I, is then cleaved by angiotensin converting enzyme (ACE) to generate the physiologically active enzyme angiotensin II. The protein is involved in maintaining blood pressure, body fluid and electrolyte homeostasis, and in the pathogenesis of essential hypertension and preeclampsia. Mutations in this gene are associated with susceptibility to essential hypertension, and can cause renal tubular dysgenesis, a severe disorder of renal tubular development. Defects in this gene have also been associated with non-familial structural atrial fibrillation, and inflammatory bowel disease. [provided by RefSeq, Nov 2019] |
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