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FABP3 (NM_004102) Human Tagged ORF Clone Lentiviral Particle
CAT＃: RC202737L4V
- LentiORF®
-
Lenti ORF particles, FABP3 (mGFP-tagged) - Human fatty acid binding protein 3, muscle and heart (mammary-derived growth inhibitor) (FABP3), 200ul, >10^7 TU/mL
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Specifications
| Product Data | |
| Product Name | FABP3 (NM_004102) Human Tagged ORF Clone Lentiviral Particle |
| Synonyms | FABP11; H-FABP; M-FABP; MDGI; O-FABP |
| Vector | pLenti-C-mGFP-P2A-Puro |
| ACCN | NM_004102 |
| ORF Size | 399 bp |
| Sequence Data |
The ORF insert of this clone is exactly the same as(RC202737).
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| OTI Disclaimer | The molecular sequence of this clone aligns with the gene accession number as a point of reference only. However, individual transcript sequences of the same gene can differ through naturally occurring variations (e.g. polymorphisms), each with its own valid existence. This clone is substantially in agreement with the reference, but a complete review of all prevailing variants is recommended prior to use. More info |
| OTI Annotation | This clone was engineered to express the complete ORF with an expression tag. Expression varies depending on the nature of the gene. |
| Reference Data | |
| RefSeq | NM_004102.3 |
| RefSeq Size | 1097 bp |
| RefSeq ORF | 402 bp |
| Locus ID | 2170 |
| Protein Pathways | PPAR signaling pathway |
| MW | 14.9 kDa |
| Gene Summary | The intracellular fatty acid-binding proteins (FABPs) belongs to a multigene family. FABPs are divided into at least three distinct types, namely the hepatic-, intestinal- and cardiac-type. They form 14-15 kDa proteins and are thought to participate in the uptake, intracellular metabolism and/or transport of long-chain fatty acids. They may also be responsible in the modulation of cell growth and proliferation. Fatty acid-binding protein 3 gene contains four exons and its function is to arrest growth of mammary epithelial cells. This gene is a candidate tumor suppressor gene for human breast cancer. Alternative splicing results in multiple transcript variants. [provided by RefSeq, Mar 2016] |
Documents
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| FAQs |
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